You need to know that there are thousands of people diagnosed HIV-positive who either stopped, or never started taking the HIV drugs and are living healthy and happy lives today, some of them for more than twenty years.

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Darin Brown Has 20 Unanswerable Questions for AIDS Inc.

1. Why have the "HIV proteins" used for antibody tests (in particular, the Western blot test) remained unchanged for 20 years (1-3), given the enormous "genetic variability/mutation rate" of HIV (4-6)?

2. Why has HIV prevalence had a constant asymmetrical geographic distribution over the past 20 years in the US (8)?

3. Why does HIV discriminate so well by race (9)? Why do organisations such as the CDC offer patently racist explanations for this data (8-10)?

4. Why have American blacks always tested HIV-positive about 5 times as often as whites, yet the ratio of AIDS cases between blacks and whites has increased 3-fold (9)?

5. Why do most individuals with low CD4 counts not develop AIDS-defining illnesses (11, 12)?

6. Why do "viral load", CD4 counts, and culturable virus have almost no correlation with each other (13, 14)?

7. Why did roughly half of all HIV-positive patients in a study have zero (undetectable) culturable virus and almost all of them have nearly undetectable culturable virus (13, 14)?

8. Why does "viral load" account for only 4% of the change in CD4 count cell loss in HIV-positive asymptomatics (15)?

9. Why do 10% of control samples from blood donors test Western blot positive (17)?

10. Why do 20-40% of ELISA-negative blood donors test Western blot "indeterminate" (18)?

11. Why are very high "viral loads" found in HIV-negative individuals? (19-21) Why is it NOT nonsensical that a laboratory test which purportedly measures the "amount of virus" requires other independent (antibody) tests to determine if virus is actually present in the first place (22)?

12. In acutely infected CEM cultures, why does cell death attributed to HIV-mediated apoptosis occur 6-7 days post-infection, while maximum virus production occurs 10-17 days post-infection (23)? The cause should always precede the effect.

13. Why do HIV test kit manufacturers now seem less convinced than ever that HIV causes AIDS (24)?

Culshaw (24) uncovered the following statements in HIV test kit disclaimers: "AIDS, AIDS-related complex and pre-AIDS are thought to be caused by HIV."; "Epidemiologic data suggest that the Acquired Immune Deficiency Syndrome (AIDS) is caused by at least two types of human immunodeficiency viruses, collectively known as HIV."; "Published data indicate a strong correlation between the acquired immune deficiency syndrome (AIDS) and a retrovirus referred to as Human Immunodeficiency Virus (HIV)." See the reference (24) for hypertext links.

14. How do CDC researchers know that detection of a certain combination of antibodies to proteins indicates infection with an exogenous retrovirus in a human, but not in a dog (25)?

15. Why has not a single chimpanzee, out of more than 250 successfully infected with HIV since 1984, developed AIDS? There is not one other human viral pathogen that cannot reproduce a similar disease in chimps.

16. Why is Pneumocystis carinii pneumonia not the most common AIDS-defining disease across all demographic and geographic spectra and why does it not occur at similar rates across demographic and geographic spectra, since it is a 100% ubiquitous latent human pathogen (27)?

17. Why do the in vivo and in vitro virus neutralizing antibodies that are present in easily assayable amounts in the blood of HIV infected people (28) not protect against AIDS if HIV is the culprit?

18. Why are exactly the same cells that HIV is said to kill in vivo not killed in vitro where productively infected cultures continue to produce 1000s of infectious particles per day for use in the various "AIDS tests" and are not protected by antibodies or "antiretroviral" drugs (29)?

19. Why are HIV and AIDS sexually equally distributed in Africa, while the ratio of male to female HIV-positive in the US is no more than 2:1, yet AIDS occurs in roughly 90% males (30)?

20. Why have improvements in "virological responses" ("viral load" and CD4 counts) to HAART not translated into decreased clinical progression to AIDS and death (31)? Among HAART patients, why do "grade 4 events" (serious or life-threatening events associated with drug toxicities) occur twice as often as "AIDS events" (32)?

BONUS: Why have all these questions been met with inadequate, if not nonexistent responses, and why have people who have raised these questions been called "irresponsible" and "dangerous to public health" (33)?